Seminario de Microbiología

Marchantia polymorpha model reveals conserved infection mechanisms in the vascular wilt fungal pathogen Fusarium oxysporum .......................Lenis Sorley Bonilla Pabon…………………The interaction between plants and microbes has been shaped by the evolution but how it has occurred is still unclear. Plants have defense mechanisms to fight back pathogens and at the same time, these pathogenic microorganisms have development strategies to enter the tissue of plants. There is an important group of pathogens causing wilt diseases in plants and are responsible for killing hundreds of crops worldwide. However, whether they can infect non-vascular plants is unknown. In this paper, the authors established a pathosystem between the root-infecting vascular wilt fungus Fusarium oxysporum (Fo) and the nonvascular plant Marchantia polymorpha (Mp). Besides, they showed that maceration and cell death of tissue of Marchantia polymorpha are caused by isolates pathogenic (Fol4287) and endophyte (Fo47) of Fusarium oxysporum. Also, they define a set of conserved fungal pathogenicity factors like cell wall remodeling enzymes and mitogen-activated protein kinases by using fungal mutants. Fungal core effectors are required for infection of Marchantia polimorpha by F. oxysporum whereas linage-specific effectors are redundant, suggesting that conserved mechanisms are employed by the fungus Fo in the infection of evolutionary distant plant lineages. Also, the authors proved that on angiosperms the fungal transition between biotrophic intercellular to necrotrophic growth requires host-specific factors, however on Marchantia polymorpha this phenomenon occurs directly……………………………https://doi.org/10.1111/nph.17909……………………………………………https://zoom.us/j/6983908886?pwd=xRlDLa9JKuEOttMI0PCJ4uJDjizFXg.1

Lugar: Auditorio Ildikó Bartnicki

Fecha: 06-11-2024

Hora: 09:00 am